Tuesday 23 June 2015

Abnormal Uterine Action



Abnormal
 Uterine
 Action
Page 1

INTRODUCTION
Normal labour is characterized
by coordinated uterine
contractions associated by
progressive dilatation of cervix
( more than 1 cm/hr) and
descent of the fetal head.

Page 2

DEFINITION
Any deviation of the normal
pattern of uterine contractions
affecting the course of labour is
designated as disorder or
abnormal uterine action.
Page 3

ETIOLOGY
Advance age of mother.
Prolonged pregnancy.
Over distension of uterus due to twins or
  hydromnios.
Psychological factors.
Contracted pelvis.
Malpresentation.
Injudicious administration of sedatives analgesics
  and oxytotics.
Premature attempt at vaginal delivery or
  instrumental vaginal delivery under light
  anesthesia.
                                              Page 4

CLASSIFICATION
A.Over-efficient uterine action
• Precipitate labour: in absence of obstruction
• Excessive contraction and retraction: in
  presence of obstruction
B.Inefficient uterine action
• Hypotonic inertia
• Hypertonic inertia
          * Colicky uterus
* Hyperactive lower uterine segment
• Constriction (contraction) ring
C.Cervical dystocia
Page 5

PRECIPITATE LABOUR
• A Labour is called precipitate when the
  combined duration of the first and
  second stage of labour is less then two
  hours.
• Labour is short as the rate of cervical
  dilatation is 5cm/hr. more in nulliparous
  women.
Page 6

ETIOLOGY
• It is more common in multiparous when
   there are:
* strong uterine contractions
* small sized baby
* roomy(no plenty of space) pelvis
* minimal soft tissue resistance
Page 7

COMPLICATIONS
Maternal-
Extensive laceration of the cervix, vagina and
perineum(to the extent of complete perineal
tear)
PPH due to uterine hypotonia
Inversion of uterus
Uterine rupture
Infection
Amniotic fluid embolism
Fetal-
Intra cranial stress and hemorrhage
Birth injuries
Page 8

MANAGEMENT
• Patient who had previous precipitate
  labour should be hospitalized before
  expected date of delivery as she is more
  prone to repeated precipitate labour.
• the uterine contractions may be
  suppressed by administering Mgso4
  during contractions.
Page 9

• Episiotomy: to avoid perineal lacerations
  and intracranial haemorrhage.
• Induction of labour by low rupture of
  membrane.
• Delivery of head should be controlled &
  careful conduction of delivery may be
  done.
Page 10

EXCESSIVE UTERINE
CONTRACTION AND
   RETRACTION
• This type of uterine contraction is
  predominantly due to obstructed labour
Page 11

Physiological Retraction Ring
• It is a line of demarcation between the upper and lower
  uterine segment present during normal labour and cannot
  usually be felt abdominally.
Pathological Retraction Ring (Bandl’s ring)
* It is the rising up retraction ring during obstructed labour
  due to marked retraction and thickening of the upper uterine
  segment while the relatively passive lower segment is
  markedly stretched and thinned to accommodate the foetus.
*The Bandl’s ring is seen and felt abdominally as a
  transverse groove that may rise to or above the symphysis
  pubis.
Page 12

Page 13

CLINICAL FEATURES
• Patient is suffering from continuous pain and
  discomfort and become restless
• Feature of exhaustion and keto acidosis are
  evident
• Abdominal palpation reveals
– Upper segment is hard and tender and lower
  segment is distended and tender
– Pathological retraction ring is placed obliquely
  between the umbilicus and symphysis pubis and
  rises upward in course of time
– Round ligament may be felt on either side
Page 14

– Fetal parts not well defined
– F.H.S. is usually absent
• Internal examination reveals
– Vagina dry and hot, discharge is offensive
– Cervix fully dilated
– Membranes are absent
– Cause of obstructed labour
Page 15

MANAGEMENT
• Internal inversion is contraindicated
• Correction of dehydration and keto
  acidosis by infusion of ringer’s solution
• Adequate pain relief
• Parenteral antibiotic
• Cesarean delivery is done majority of the
  cases
Page 16

 UTERINE INERTIA
(hypotonic activity)
• Uterine inertia is a common type of disordered
  uterine contractions and may complicate any
  stage of labour.
• The intensity of uterine contraction is diminished
  duration is shortened, good relaxation in between
  contractions and the intervals are increased.
• General pattern of uterine contraction of labour is
  maintained but intrauterine pressure during
  contraction hardly rises above 25 mm
Page 17

• Patient feels less pain during uterine
  contractions.
• Less hardening of the uterus during contraction
• Uterus become relaxed after the contraction.
• Internal examination reveals:-
      a.Over dilatation of cervix.
      b.Associated presence of contracted pelvis,
       malposition, deflexed head or
       Malpresentation.
      c.Membranes usually remains intact.
Page 18
DIAGNOSIS

Effect on the Mother and
          Fetus
• Mother: maternal exhaustion
• Fetus: fetal distress.
Page 19

MANAGEMENT
Contemplating vaginal delivery
A.General measures:
1.To keep up the moral of the patient.
2.Posture of the patient is changed (left lateral position)
  supine position is avoided.
3.To empty the bladder by catheterization if needed.
4.To maintain the hydration by infusion of RL solution.
5. Adequate pain relief by intramuscular pethedine 100mg
Page 20

B. Active measures:
• acceleration of uterine contraction by lower
  rupture of membrane followed by Oxytocin
  drip.
• Oxytocin is to be continued till 1 hr after
  delivery.
• if cervical dilatation remains unsatisfactory
  and fetal distress appears, caesarian
           section should be done.
Page 21

HYPERTONIC UTERINE INERTIA
 (Inco-ordinate Uterine Action)
• the hypertonic state of uterus arises
  from any of the conditions such as
  spastic lower uterine segment ,colicky
  uterus , contraction , constriction ring
  or generalized uterine contraction of
  the uterus and all the states are
  collectively called incordinate uterine
  action
Page 22

TYPES
• Colicky uterus: incoordination of the
  different parts of the uterus in
  contractions.
• Hyperactive lower uterine segment: so
  the dominance of the upper segment is
  lost.
Page 23

CLINICAL FEATURES
The condition is more common in primigravidae and
 characterised by:
Labour is prolonged.
• Uterine contractions are irregular and more painful. The
  pain is felt before and throughout the contractions with
  marked low backache often in occipito-posterior position.
• High resting intrauterine pressure in between uterine
  contractions detected by tocography (normal value is 5-10
  mmHg).
• Slow cervical dilatation .
• Premature rupture of membranes.
• Foetal and maternal distress.
Page 24

MANAGEMENT
A. General measures: as hypotonic inertia.
B. Medical measures:
– Analgesic and antispasmodic as pethidine.
– Epidural analgesia may be of good benefit.
C. Caesarean section is indicated in:
– Failure of the previous methods.
– Disproportion.
– Foetal distress before full cervical dilatation.
Page 25

   CONSTRICTION
(CONTRACTION) RING
• It is a persistent localized annular spasm
  of the circular uterine muscles.
• It occurs at any part of the uterus but
  usually at junction of the upper and lower
  uterine segments.
• It can occur at the 1st, 2nd or 3 rd stage
  of labour.
Page 26

ETIOLOGY
Unknown but the predisposing factors are:
* Malpresentations and malpositions.
* Clumsy intrauterine manipulations under
   light anaesthesia.
* Improper use of oxytocin e.g.
       > use of oxytocin in hypertonic inertia.
       >IM injection of oxytocin.
Page 27

DIAGNOSIS
• The condition is more common in
  primigravidae and frequently preceded by
  colicky uterus.
• The exact diagnosis is achieved only by
  feeling the ring with a hand introduced into
  the uterine cavity.
Page 28

COMPLICATION
• Prolonged 1st stage: if the ring occurs at
  the level of the internal os.
• Prolonged 2nd stage: if the ring occurs
  around the foetal neck.
• Retained placenta and postpartum
  haemorrhage: if the ring occurs in the 3rd
  stage (hour- glass contraction).
Page 29

Pathological Retraction
         Ring
Occurs in prolonged 2nd stage.
Always between upper and lower
uterine segments.
Rises up.
Felt and seen abdominally.
The uterus is tonically retracted, tender
and the foetal parts cannot be felt.
Constriction Ring
Occurs in the 1st, 2nd or 3rd stage.
At any level of the uterus.
Does not change its position.
Felt only vaginally.
The uterus is not tonically retracted and
the foetal parts can be felt.
Maternal distress and foetal distress or
death.
Relieved only by delivery of the foetus.
Maternal and foetal distress may not be
present.
May be relieved by anaesthetics or
antispasmodics.
Page 30
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MANAGEMENT
• In the 1st stage: Pethidine may be of
  benefit.
• In the 2nd stage: Deep general
  anaesthesia and amyl nitrite inhalation are
  given to relax the constriction ring:
• In the 3rd stage: Deep general
  anaesthesia and amyl nitrite inhalation are
  given followed by manual removal of the
  placenta.
Page 31

CERVICAL DYSTOCIA
• Failure of the cervix to dilate within a
  reasonable time in spite of good
  regular uterine contractions.
Page 32

TYPES
A. Functional (primary):
> In spite of the absence of any organic
  lesion and the well effacement of the
  cervix, the external os fails to dilate.
> This may be due to lack of softening of the
  cervix during pregnancy or cervical spasm
  resulted from overactive sympathetic tone
Page 33

B. Organic (secondary) due to:
> Cervical scarring related to previous
  amputation, cone biopsy, extensive
  cauterisation or obstetric trauma.
> Organic lesions as cervical myoma or
  carcinoma.
Page 34

COMPLICATIONS
• Annular detachment of the cervix:
  surprisingly the bleeding from the cervix is
  minimal because of fibrosis and avascular
  pressure necrosis leading to thrombosis of
  the vessels before detachment.
• Rupture uterus.
• Postpartum hemorrhage: particularly if
  cervical laceration extends upwards
  tearing the main uterine vessels.
Page 35

MANAGEMENT
A. Organic dystocia:
> Caesarean section is the management of
  choice.
B.Functional dystocia:
Pethidine and antispasmodics: may be
  effective
Caesarean section: if medical treatment
  fails or foetal distress developed.
Page 36

PREMATURE RUPTURE OF
MEMBRANES
(PROM)
Page 37

Definition
PROM is defined as the rupture
of the chorioamniotic membrane
before the onset of labor
Page 38

Incidence
PROM occurs in about
10%~15% of all delivery
PROM is associated with 10%
of term pregnancy
Page 39

Cause of PROM
Trauma (abdominal striked intensely)
Sexual intercourse(particularly in the late
 gestational weeks)
Vaginal infection
  due to
 bacteria virus TOXO CMV HPV and
 HSV,et al
Smoking
Page 40

Other :
Prior PROM
A short cervical length
Prior preterm delivery
Bleeding in early pregancy
Page 41

Mothers:
(1) Infection : intrauterine
                puerperal
(2) Placental abruption
(3) Preterm delivery
Page 42

     Infants:
(1) Preterm Baby and their Complications :
    (RDS / Fetal and Neurologic dysfunction
     Intracranial hemorrhage)
(2) neonatal pneumonia sepsis
(3) Pulmonary hypoplasia and fetal
  compression syndrone
(4) Prolapse or compression of umbilical cord
(5) Abruptio placenta
Page 43

Clinical manifestation
(1) Fluid passing through the vagina
  suddenly, and then small amounts of fluid
  flow through the vagina intermitently,
  particularly when the increased of
  abdorminal pressure (cough,sneeze,et al)
Page 44

(2) Free flowing amniotic fluid
(3) Fever / heart rate of mother and
    infants / WBC and CRP ↑/ Uterine
    tenderness on palpation
Page 45

Vaginal speculum exam.:
    leakage of amniotic fluid
(2) PH determination of vaginal fluid
(3) The “fern” test
(4) Aminoscopy
Diagnosis
(1)
Page 46

Management
PROM at term:
(1) Awaiting the onset of spontaneous labor for 12-24h
(2) Termination of pregnancy after 24 hours
PROM before term:
  Termination of pregnancy
(1) Evidence of fetal pulmonary maturation
(2) Evidence of inturine infection
Page 47

Expectant therapy
Indication :
(1) Evidence of fetal pulmonary inmaturation
(2) Without evidence of inturine infection
Management:
(1)To enhance fetal pulmonary maturation
(2) Antibiotic
(3) Tocolysis



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